Researchers reveal that the loss of mucin-type O-glycosylation in the brain endothelial glycocalyx weakens the blood-brain ...

Once inside the CNS, the bacteria multiply and induce inflammation of the subarachnoid and ventrical spaces with associated pathophysiologic alterations such as increased BBB permeability and ...

Significant progress has been made in identifying molecular mechanisms that contribute to bacterial–BBB interaction and signaling during the progression of CNS disease. Identification of common ...