The delicate balance between cell death and regeneration is crucial for human health and longevity. Most mechanisms involved ...

Fat isn’t just a guilty pleasure — it’s cancer’s secret weapon. New research from UT Southwestern Medical Center is shedding ...

A recent study involving researchers from the University of Basel reveals that slowing down the intracellular transport of ...

This is a useful study in the role of CHI3L1 in Kupffer cells, the macrophages of the liver, showing that CHI3L1 alters glucose regulation in obesity. Specifically, Chi3l1 protects glucose-dependent ...

In a remarkable twist of science, researchers have transformed a fungus long associated with death into a potential weapon ...

While this research is still in its early stages, it opens the door for new drug development. Scientists now hope to create ...

This study unravels that ferroptosis, an iron-mediated cell death characterized by lipid oxidation, can be initiated in lysosomes. Ferroptosis has been gathering an increased interest in cancer ...

The team discovered that peroxidation of lysosomes plays a key role in the execution of ferroptosis-mediated cell death. Additionally, they found that lysosomal lipid peroxidation causes the membrane ...

Ferroptosis, a form of programmed cell death mediated by iron, has been a focus for its potential in cancer therapies. Now, researchers have discovered that lysosomal lipid peroxidation plays a ...

For example, in humans, white blood cells die between 60 and 86 hours after organismal death. In mice, skeletal muscle cells can be regrown after 14 days postmortem, ...

Programmed cell death, ... The cell breaks down its own worn components by transporting them to lysosomes, which are like small stomachs in the cell, where they are consumed. Autophagy can function ...